ERα36–GPER1 Collaboration Inhibits TLR4/NFκB-Induced Pro-Inflammatory Activity in Breast Cancer Cells

Inflammation is important for the initiation and progression of breast cancer. We have previously reported that in monocytes, estrogen regulates TLR4/NFκB-mediated inflammation via the interaction of the Erα isoform ERα36 with GPER1. We therefore investigated whether a similar mechanism is present i...

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Bibliografiska uppgifter
I publikationen:Int J Mol Sci
Huvudupphovsmän: Notas, George, Panagiotopoulos, Athanasios, Vamvoukaki, Rodanthi, Kalyvianaki, Konstantina, Kiagiadaki, Foteini, Deli, Alexandra, Kampa, Marilena, Castanas, Elias
Materialtyp: Artigo
Språk:Inglês
Publicerad: MDPI 2021
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC8303269/
https://ncbi.nlm.nih.gov/pubmed/34299224
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms22147603
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