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Increased Glutamine in Patients Undergoing Long-term Treatment for Schizophrenia: A Proton Magnetic Resonance Spectroscopy Study at 3 T

IMPORTANCE: The N-methyl-d-aspartic acid receptor hypofunction model of schizophrenia predicts a paradoxical increase in synaptic glutamate release. In vivo measurement of glutamatergic neurotransmission in humans is challenging, but glutamine, the principal metabolite of synaptic glutamate, can be...

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Detalhes bibliográficos
Publicado no:JAMA Psychiatry
Main Authors: Bustillo, Juan R., Chen, Hongji, Jones, Thomas, Lemke, Nicholas, Abbott, Christopher, Qualls, Clifford, Canive, Jose, Gasparovic, Charles
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC8185982/
https://ncbi.nlm.nih.gov/pubmed/24402128
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1001/jamapsychiatry.2013.3939
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