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Diabetic Ketoacidosis and Dysregulation of Proglucagon Family of Molecules
OBJECTIVES: An increasing body of evidence supports a significant contributory role for pancreatic α-cell dysfunction in the pathophysiology of diabetic ketoacidosis (DKA). In normal physiology, high glucose levels stimulate insulin and inhibit glucagon secretion, while low glucose stimulates glucag...
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| Publicado no: | Curr Dev Nutr |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2021
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8182090/ https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cdn/nzab047_020 |
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