Nitric oxide down-regulates voltage-gated Na(+) channel in cardiomyocytes possibly through S-nitrosylation-mediated signaling

Nitric oxide (NO) is produced from endothelial cells and cardiomyocytes composing the myocardium and benefits cardiac function through both vascular-dependent and—independent effects. This study was purposed to investigate the possible adverse effect of NO focusing on the voltage-gated Na(+) channel...

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Detalhes bibliográficos
Publicado no:Sci Rep
Main Authors: Wang, Pu, Wei, Mengyan, Zhu, Xiufang, Liu, Yangong, Yoshimura, Kenshi, Zheng, Mingqi, Liu, Gang, Kume, Shinichiro, Morishima, Masaki, Kurokawa, Tatsuki, Ono, Katsushige
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2021
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC8163867/
https://ncbi.nlm.nih.gov/pubmed/34050231
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-021-90840-0
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