טוען...
Nitric oxide down-regulates voltage-gated Na(+) channel in cardiomyocytes possibly through S-nitrosylation-mediated signaling
Nitric oxide (NO) is produced from endothelial cells and cardiomyocytes composing the myocardium and benefits cardiac function through both vascular-dependent and—independent effects. This study was purposed to investigate the possible adverse effect of NO focusing on the voltage-gated Na(+) channel...
שמור ב:
| הוצא לאור ב: | Sci Rep |
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| Main Authors: | , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Nature Publishing Group UK
2021
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8163867/ https://ncbi.nlm.nih.gov/pubmed/34050231 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-021-90840-0 |
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