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Regulation of Mitochondrial Function by Epac2 Contributes to Acute Inflammatory Hyperalgesia
G(αs)-coupled receptors signaling through cAMP provide a key mechanism for the sensitization of nociceptive sensory neurons, and the cAMP effector Epac has been implicated in the transition from acute to chronic pain. Epac exerts its effects through Rap1 and protein kinase C (PKC). To identify targe...
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| Udgivet i: | J Neurosci |
|---|---|
| Main Authors: | , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Society for Neuroscience
2021
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8018894/ https://ncbi.nlm.nih.gov/pubmed/33593853 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2368-20.2021 |
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