Deletion of the Thrombin Proteolytic Site in Neurofascin 155 Causes Disruption of Nodal and Paranodal Organization

Lignende værker

• Novel forms of neurofascin 155 in the central nervous system: alterations in paranodal disruption models and multiple sclerosis
  af: Pomicter, Anthony D., et al.
  Udgivet: (2010)
• Does Paranode Formation and Maintenance Require Partitioning of Neurofascin 155 into Lipid Rafts?
  af: Schafer, Dorothy P., et al.
  Udgivet: (2004)
• Anti–neurofascin-155 IgG4 antibodies prevent paranodal complex formation in vivo
  af: Manso, Constance, et al.
  Udgivet: (2019)
• mTORC1 Activation by Loss of Tsc1 in Myelinating Glia Causes Downregulation of Quaking and Neurofascin 155 Leading to Paranodal Domain Disorganization
  af: Shi, Qian, et al.
  Udgivet: (2018)
• Focal loss of the paranodal domain protein Neurofascin155 in the internal capsule impairs cortically induced muscle activity in vivo
  af: Kunisawa, Kazuo, et al.
  Udgivet: (2020)