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Maternal selenium deficiency in mice promotes sex‐specific changes to urine flow and renal expression of mitochondrial proteins in adult offspring

Selenium deficiency during pregnancy can impair fetal development and predispose offspring to thyroid dysfunction. Given that key selenoproteins are highly expressed in the kidney and that poor thyroid health can lead to kidney disease, it is likely that kidney function may be impaired in offspring...

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Detalhes bibliográficos
Publicado no:Physiol Rep
Main Authors: Neal, Elliott S., Hofstee, Pierre, Askew, Montana R., Kent, Nykola L., Bartho, Lucy A., Perkins, Anthony V., Cuffe, James S. M.
Formato: Artigo
Idioma:Inglês
Publicado em: John Wiley and Sons Inc. 2021
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7995548/
https://ncbi.nlm.nih.gov/pubmed/33769708
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14785
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