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Astrocytes respond to a neurotoxic Aβ fragment with state-dependent Ca(2+) alteration and multiphasic transmitter release
Excessive amounts of amyloid β (Aβ) peptide have been suggested to dysregulate synaptic transmission in Alzheimer’s disease (AD). As a major type of glial cell in the mammalian brain, astrocytes regulate neuronal function and undergo activity alterations upon Aβ exposure. Yet the mechanistic steps u...
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| Publicado no: | Acta Neuropathol Commun |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2021
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7968286/ https://ncbi.nlm.nih.gov/pubmed/33726852 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-021-01146-1 |
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