Peptide Szeto-Schiller 31 ameliorates doxorubicin-induced cardiotoxicity by inhibiting the activation of the p38 MAPK signaling pathway

Oxidative stress serves a key role in doxorubicin (DOX)-induced cardiotoxicity. The peptide Szeto-Schiller (SS)31 is an efficacious antioxidant with the capacity to reduce mitochondrial reactive oxygen species (ROS) levels and scavenge free radicals. Although SS31 is involved in the pathophysiologic...

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Detaylı Bibliyografya
Yayımlandı:Int J Mol Med
Asıl Yazarlar: Zhang, Li, Feng, Mengwen, Wang, Xuejun, Zhang, Hao, Ding, Jingjing, Cheng, Zijie, Qian, Lingmei
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: D.A. Spandidos 2021
Konular:
Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC7914074/
https://ncbi.nlm.nih.gov/pubmed/33649779
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/ijmm.2021.4896
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