Peptide Szeto-Schiller 31 ameliorates doxorubicin-induced cardiotoxicity by inhibiting the activation of the p38 MAPK signaling pathway

Oxidative stress serves a key role in doxorubicin (DOX)-induced cardiotoxicity. The peptide Szeto-Schiller (SS)31 is an efficacious antioxidant with the capacity to reduce mitochondrial reactive oxygen species (ROS) levels and scavenge free radicals. Although SS31 is involved in the pathophysiologic...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Cyhoeddwyd yn:Int J Mol Med
Prif Awduron: Zhang, Li, Feng, Mengwen, Wang, Xuejun, Zhang, Hao, Ding, Jingjing, Cheng, Zijie, Qian, Lingmei
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: D.A. Spandidos 2021
Pynciau:
Articles
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC7914074/
https://ncbi.nlm.nih.gov/pubmed/33649779
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/ijmm.2021.4896
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