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DNA- and telomere-damage does not limit lifespan: evidence from rapamycin
Failure of rapamycin to extend lifespan in DNA repair mutant and telomerase-knockout mice, while extending lifespan in normal mice, indicates that neither DNA damage nor telomere shortening limits normal lifespan or causes normal aging.
Bewaard in:
| Gepubliceerd in: | Aging (Albany NY) |
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| Hoofdauteur: | |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Impact Journals
2021
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7906135/ https://ncbi.nlm.nih.gov/pubmed/33578394 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/aging.202674 |
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