CaMKII exacerbates heart failure progression by activating class I HDACs

BACKGROUND: Persistent cardiac Ca(2+)/calmodulin dependent Kinase II (CaMKII) activation plays an essential role in heart failure development. However, the molecular mechanisms underlying CaMKII induced heart failure progression remains incompletely understood. Histone deacetylases (HDACs) are criti...

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Bibliografiset tiedot
Julkaisussa:J Mol Cell Cardiol
Päätekijät: Zhang, Manling, Yang, Xue, Zimmerman, Raymond J., Wang, Qin, Ross, Mark A., Granger, Jonathan M., Luczak, Elizabeth D., Bedja, Djahida, Jiang, Hong, Feng, Ning
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2020
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7736490/
https://ncbi.nlm.nih.gov/pubmed/32971072
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2020.09.007
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