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Peroxiredoxin2 (Prdx2) Reduces Oxidative Stress and Apoptosis of Myocardial Cells Induced by Acute Myocardial Infarction by Inhibiting the TLR4/Nuclear Factor kappa B (NF-κB) Signaling Pathway

BACKGROUND: Peroxiredoxin2 (Prdx2) is an endogenous peroxidase and has been found to reduce the oxidative burden in cells and thereby reduce cell damage and apoptosis. Therefore, the purpose of this study was to investigate the effect of Prdx2 on the oxidative level and apoptosis of myocardial cells...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Med Sci Monit
Κύριοι συγγραφείς: Li, Hangshun, Yang, Haina, Wang, Dandan, Zhang, Lixin, Ma, Tao
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: International Scientific Literature, Inc. 2020
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7722772/
https://ncbi.nlm.nih.gov/pubmed/33268762
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12659/MSM.926281
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