Hyperphosphorylation of fetal liver IGFBP-1 precedes slowing of fetal growth in nutrient-restricted baboons and may be a mechanism underlying IUGR

In cultured fetal liver cells, insulin-like growth factor (IGF) binding protein (IGFBP)-1 hyperphosphorylation in response to hypoxia and amino acid deprivation is mediated by inhibition of mechanistic target of rapamycin (mTOR) and activation of amino acid response (AAR) signaling and casein kinase...

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Detalhes bibliográficos
Publicado no:Am J Physiol Endocrinol Metab
Main Authors: Kakadia, Jenica H., Jain, Bhawani B., Biggar, Kyle, Sutherland, Austen, Nygard, Karen, Li, Cun, Nathanielsz, Peter W., Jansson, Thomas, Gupta, Madhulika B.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2020
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7642856/
https://ncbi.nlm.nih.gov/pubmed/32744097
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00220.2020
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