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SLE: Novel Postulates for Therapeutic Options
Genetic deficiency in C1q is a strong susceptibility factor for systemic lupus erythematosus (SLE). There are two major hypotheses that potentially explain the role of C1q in SLE. The first postulates that C1q deficiency abrogates apoptotic cell clearance, leading to persistently high loads of poten...
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| Udgivet i: | Front Immunol |
|---|---|
| Main Authors: | , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Frontiers Media S.A.
2020
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7575694/ https://ncbi.nlm.nih.gov/pubmed/33117397 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2020.583853 |
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