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β-catenin regulates muscle glucose transport via actin remodelling and M-cadherin binding
OBJECTIVE: Skeletal muscle glucose disposal following a meal is mediated through insulin-stimulated movement of the GLUT4-containing vesicles to the cell surface. The highly conserved scaffold-protein β-catenin is an emerging regulator of vesicle trafficking in other tissues. Here, we investigated t...
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| Опубликовано в: : | Mol Metab |
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| Главные авторы: | , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
Elsevier
2020
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7568189/ https://ncbi.nlm.nih.gov/pubmed/33011305 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molmet.2020.101091 |
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