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GPR68 deletion impairs hippocampal long-term potentiation and passive avoidance behavior
Increased neural activities reduced pH at the synaptic cleft and interstitial spaces. Recent studies have shown that protons function as a neurotransmitter. However, it remains unclear whether protons signal through a metabotropic receptor to regulate synaptic function. Here, we showed that GPR68, a...
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| Vydáno v: | Mol Brain |
|---|---|
| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
BioMed Central
2020
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7526169/ https://ncbi.nlm.nih.gov/pubmed/32993733 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-020-00672-8 |
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