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Neutralization of Lipocalin-2 Diminishes Stroke-Reperfusion Injury
Oxidative stress is a key contributor to the pathogenesis of stroke-reperfusion injury. Neuroinflammatory peptides released after ischemic stroke mediate reperfusion injury. Previous studies, including ours, have shown that lipocalin-2 (LCN2) is secreted in response to cerebral ischemia to promote r...
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| Izdano u: | Int J Mol Sci |
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| Glavni autori: | , , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
MDPI
2020
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7503651/ https://ncbi.nlm.nih.gov/pubmed/32872405 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms21176253 |
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