PRL-3 facilitates Hepatocellular Carcinoma progression by co-amplifying with and activating FAK

Background: In addition to protein tyrosine kinases, accumulating evidence has shown that protein tyrosine phosphatases (PTPs) are suitable therapeutic targets in cancer. PRL-3 is a PTP member that has been well studied in many malignant tumours. The goal of the present study was to elucidate the ro...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
الحاوية / القاعدة:Theranostics
المؤلفون الرئيسيون: Zhou, Qiming, Zhou, Qianlei, Liu, Qinghua, He, Zhanghai, Yan, Yongcong, Lin, Jianhong, Chen, Zheng, He, Chuanchao, Mao, Kai, Wang, Jie, Zhou, Zhenyu, Xiao, Zhiyu, Zhang, Jianlong
التنسيق: Artigo
اللغة:Inglês
منشور في: Ivyspring International Publisher 2020
الموضوعات:
Research Paper
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC7481414/
https://ncbi.nlm.nih.gov/pubmed/32929353
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.42069
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