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IL-36 receptor antagonist deficiency resulted in delayed wound healing due to excessive recruitment of immune cells
Loss-of-function homozygous or compound heterozygous mutations in IL36RN, which encodes interleukin-36 receptor antagonist (IL-36Ra), have been implicated in the pathogenesis of various skin disorders. Previous findings showed that IL-36γ promoted wound healing in mice; however, the pathogenic role...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7479622/ https://ncbi.nlm.nih.gov/pubmed/32901055 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-71256-8 |
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