Testicular blood supply is altered in the 41,XX(Y)* Klinefelter syndrome mouse model

Hypergonadotropic hypogonadism is a major feature of Klinefelter syndrome (KS), assumed to be caused by testicular hormone resistance. It was previously shown that intratesticular testosterone levels in vivo and Leydig cell function in vitro seem to be normal indicating other functional constraints....

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Detalhes bibliográficos
Publicado no:Sci Rep
Main Authors: Wistuba, Joachim, Beumer, Cristin, Warmeling, Ann-Sophie, Sandhowe-Klaverkamp, Reinhild, Stypmann, Jörg, Kuhlmann, Michael, Holtmeier, Richard, Damm, Oliver S., Tüttelmann, Frank, Gromoll, Jörg
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2020
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7462989/
https://ncbi.nlm.nih.gov/pubmed/32873847
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-71377-0
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