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Inhibiting the PGE(2) Receptor EP2 Mitigates Excitotoxicity and Ischemic Injury
[Image: see text] Prostaglandin E2 (PGE(2)) is elevated in the brain by excitotoxic insults and, in turn, aggravates the neurotoxicity mainly through acting on its Gα(s)-coupled receptor EP2, inspiring a therapeutic strategy of targeting this key proinflammatory pathway. Herein, we investigated the...
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| Publicado en: | ACS Pharmacol Transl Sci |
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| Autores principales: | , , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American
Chemical Society
2020
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| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7432651/ https://ncbi.nlm.nih.gov/pubmed/32832866 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/acsptsci.0c00040 |
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