Inhibition of PAR-2 attenuates neuroinflammation and improves short-term neurocognitive functions via ERK1/2 signaling following asphyxia-induced cardiac arrest in rats

OBJECTIVE: Global cerebral ischemia induced neuroinflammation causes neurofunctional impairment following cardiac arrest. Previous studies have demonstrated that the activation of protease activated receptor-2 (PAR-2) contributes to neuroinflammation. In the present study, we aimed to determine the...

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Gepubliceerd in:Shock
Hoofdauteurs: Ocak, Umut, Ocak, Pinar Eser, Huang, Lei, Zuo, Gang, Yan, Jun, Hu, Xin, Song, Zhijun, Zhang, John H.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2020
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7415486/
https://ncbi.nlm.nih.gov/pubmed/32028357
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/SHK.0000000000001516
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