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Glucocorticoid Exposure Induces Preeclampsia via DampeningLipoxin A(4), an Endogenous Anti-Inflammatory and Proresolving Mediator
The pathogenesis of preeclampsia (PE) involves several pathophysiological processes that may be affected by glucocorticoid (GC). We confirmed previously that GC exposure could result in PE, while PE is linked to a deficiency of lipoxin A(4) (LXA(4)), an endogenous dual anti-inflammatory and proresol...
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| Foilsithe in: | Front Pharmacol |
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| Main Authors: | , , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Frontiers Media S.A.
2020
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7399346/ https://ncbi.nlm.nih.gov/pubmed/32848749 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphar.2020.01131 |
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