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Extracellular gp96 is a crucial mediator for driving immune hyperactivation and liver damage
Liver failure leads to the massive necrosis of hepatocytes, releasing large amounts of intracellular components including damage-associated molecular patterns (DAMPs). We found that extracellular gp96 levels in serum were elevated in patients with chronic hepatitis B infection (CHB) and acute-on-chr...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7387550/ https://ncbi.nlm.nih.gov/pubmed/32724151 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-69517-7 |
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