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Targeting mitochondrial fusion and fission proteins for cardioprotection
New treatments are needed to protect the myocardium against the detrimental effects of acute ischaemia/reperfusion (IR) injury following an acute myocardial infarction (AMI), in order to limit myocardial infarct (MI) size, preserve cardiac function and prevent the onset of heart failure (HF). Given...
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| Foilsithe in: | J Cell Mol Med |
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| Main Authors: | , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
John Wiley and Sons Inc.
2020
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7299693/ https://ncbi.nlm.nih.gov/pubmed/32406208 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.15384 |
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