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BGP-15 Protects Mitochondria in Acute, Acetaminophen Overdose Induced Liver Injury
Acetaminophen (APAP) induced hepatotoxicity involves activation of c-Jun amino-terminal kinase (JNK), mitochondrial damage and ER stress. BGP-15, a hydroximic acid derivative, has been reported to have hepatoprotective effects in APAP overdose induced liver damage. Effect of BGP-15 was further inves...
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| Publicado no: | Pathol Oncol Res |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Springer Netherlands
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7297855/ https://ncbi.nlm.nih.gov/pubmed/31705481 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12253-019-00721-1 |
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