Central AT1 receptor signaling by circulating angiotensin II is permissive to acute intermittent hypoxia-induced sympathetic neuroplasticity

Lignende værker

• Hypothalamic PVN contributes to acute intermittent hypoxia-induced sympathetic but not phrenic long-term facilitation
  af: Blackburn, Megan B., et al.
  Udgivet: (2018)
• Activation of the hypothalamic paraventricular nucleus by acute intermittent hypoxia: Implications for sympathetic long-term facilitation neuroplasticity
  af: Maruyama, Nadia Oliveira, et al.
  Udgivet: (2018)
• Effect of Chronic Intermittent Hypoxia on Angiotensin II Receptors in the Central Nervous System
  af: Morgan, Barbara J., et al.
  Udgivet: (2018)
• Chronic intermittent hypoxia increases sympathetic control of blood pressure: role of neuronal activity in the hypothalamic paraventricular nucleus
  af: Sharpe, Amanda L., et al.
  Udgivet: (2013)
• Neurogenic mechanisms underlying the rapid onset of sympathetic responses to intermittent hypoxia
  af: Mifflin, Steve, et al.
  Udgivet: (2015)