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RECK isoforms differentially regulate fibroblast migration by modulating tubulin post- translational modifications
Cell migration is essential for proper development and the defense against pathogens. Our previous work detailed a pathway of REversion-inducing-Cysteine-rich protein with Kazal motifs (RECK) isoform-mediated invasion in which a shorter RECK protein competes with MMP9 for interaction with the canoni...
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| 出版年: | Biochem Biophys Res Commun |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7217981/ https://ncbi.nlm.nih.gov/pubmed/30704758 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2019.01.063 |
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