Mycobacterium tuberculosis Heat-Shock Protein 16.3 Induces Macrophage M2 Polarization Through CCRL2/CX3CR1

Mycobacterium tuberculosis, the pathogen of tuberculosis (TB), can survive in host macrophages and induce macrophages to M2 phenotype might result in latent MTB infection. During the latent phase, the expression of MTB heat-shock protein 16.3 (Hsp16.3) is markedly increased among most of bacterial p...

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Publicat a:Inflammation
Autors principals: Zhang, Yanhao, Li, Shanshan, Liu, Qianyi, Long, Ruiying, Feng, Jihong, Qin, Huan, Li, Mao, Liu, Liping, Luo, Junmin
Format: Artigo
Idioma:Inglês
Publicat: Springer US 2019
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Original Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7170987/
https://ncbi.nlm.nih.gov/pubmed/31748849
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10753-019-01132-9
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