Neutrophil-derived S100A8/A9 amplify granulopoiesis following myocardial infarction

BACKGROUND: Myocardial infarction (MI) triggers myelopoiesis resulting in heightened production of neutrophils. However, the mechanisms that sustain their production and recruitment to the injured heart are unclear. METHODS: Using a mouse model of the permanent ligation of the left anterior descendi...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Circulation
Päätekijät: Sreejit, Gopalkrishna, Abdel-Latif, Ahmed, Athmanathan, Baskaran, Annabathula, Rahul, Dhyani, Ashish, Noothi, Sunil K, Quaife-Ryan, Gregory A., Al-Sharea, Annas, Pernes, Gerard, Dragoljevic, Dragana, Lal, Hind, Schroder, Kate, Hanaoka, Beatriz Y, Raman, Chander, Grant, Maria B, Hudson, James E., Smyth, Susan, Porrello, Enzo R., Murphy, Andrew J., Nagareddy, Prabhakara R.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2020
Aiheet:
Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7122461/
https://ncbi.nlm.nih.gov/pubmed/31941367
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCULATIONAHA.119.043833
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