Botulinum toxin type A promotes microglial M2 polarization and suppresses chronic constriction injury-induced neuropathic pain through the P2X7 receptor

BACKGROUND: Switching microglial polarization from the pro-inflammatory M1 phenotype to the anti-inflammatory M2 phenotype represents a novel therapeutic strategy for neuropathic pain (NP). This study aims to investigate whether botulinum toxin type A (BTX-A) regulates microglial M1/M2 polarization...

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Udgivet i:Cell Biosci
Main Authors: Gui, Xianwei, Wang, Hansen, Wu, Lanxiang, Tian, Sheng, Wang, Xuan, Zheng, Heqing, Wu, Wei
Format: Artigo
Sprog:Inglês
Udgivet: BioMed Central 2020
Fag:
Research
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7092425/
https://ncbi.nlm.nih.gov/pubmed/32211150
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13578-020-00405-3
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