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Antisense oligonucleotides targeting Notch2 ameliorate the osteopenic phenotype in a mouse model of Hajdu-Cheney syndrome

Notch receptors play critical roles in cell-fate decisions and in the regulation of skeletal development and bone remodeling. Gain–of–function NOTCH2 mutations can cause Hajdu-Cheney syndrome, an untreatable disease characterized by osteoporosis and fractures, craniofacial developmental abnormalitie...

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Detalles Bibliográficos
Publicado en:J Biol Chem
Autores principales: Canalis, Ernesto, Grossman, Tamar R., Carrer, Michele, Schilling, Lauren, Yu, Jungeun
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Biochemistry and Molecular Biology 2020
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC7086019/
https://ncbi.nlm.nih.gov/pubmed/31992595
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA119.011440
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