In utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure exacerbates urinary dysfunction in hormone-treated C57BL/6J mice through a non-malignant mechanism involving proteomic changes in the prostate that differ from those elicited by testosterone and estradiol

A recent study directed new focus on the fetal and neonatal environment as a risk factor for urinary dysfunction in aging males. Male mice were exposed in utero and via lactation (IUL) to the persistent environmental contaminant 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and then administered slow-r...

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Detalhes bibliográficos
Publicado no:Am J Clin Exp Urol
Main Authors: Turco, Anne E, Thomas, Samuel, Crawford, LaTasha K, Tang, Weiping, Peterson, Richard E, Li, Lingjun, Ricke, William A, Vezina, Chad M
Formato: Artigo
Idioma:Inglês
Publicado em: e-Century Publishing Corporation 2020
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Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7076297/
https://ncbi.nlm.nih.gov/pubmed/32211455
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