Fibroblast growth factor-2-induced cardioprotection against myocardial infarction occurs via the interplay between nitric oxide, protein kinase signaling, and ATP-sensitive potassium channels

Fibroblast growth factor-2 (FGF2) protects the heart from ischemia–reperfusion (I-R) injury via a vast network of protein kinases. In the heart, downstream effectors of these FGF2-triggered signals have not yet been identified. It is hypothesized that nitric oxide (NO) signaling and ATP-sensitive po...

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Vydáno v:Growth Factors
Hlavní autoři: MANNING, JANET R., CARPENTER, GREGORY, PORTER, DARIUS R., HOUSE, STACEY L., PIETRAS, DANIEL A., DOETSCHMAN, THOMAS, SCHULTZ, JO EL J.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2012
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC7041406/
https://ncbi.nlm.nih.gov/pubmed/22304432
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3109/08977194.2012.656759
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