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CTRP3 induces an intermediate switch of CD14(++)CD16(+) monocyte subset with anti-inflammatory phenotype
Acute myocardial infarction (AMI) evokes a temporally coordinated immune response, in which monocytes are critically involved in the clearance of cell debris; however, excessive inflammation induced by the classical sub-population of monocytes frequently limits the endogenous reparative process. In...
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| Pubblicato in: | Exp Ther Med |
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| Autori principali: | , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
D.A. Spandidos
2020
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7027268/ https://ncbi.nlm.nih.gov/pubmed/32104290 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/etm.2020.8467 |
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