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Zn(2+) entry through the mitochondrial calcium uniporter is a critical contributor to mitochondrial dysfunction and neurodegeneration
Excitotoxic Ca(2+) accumulation contributes to ischemic neurodegeneration, and Ca(2+) can enter the mitochondria through the mitochondrial calcium uniporter (MCU) to promote mitochondrial dysfunction. Yet, Ca(2+)-targeted therapies have met limited success. A growing body of evidence has highlighted...
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| 出版年: | Exp Neurol |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6957126/ https://ncbi.nlm.nih.gov/pubmed/31881218 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2019.113161 |
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