טוען...
Interleukin‐38 alleviates cardiac remodelling after myocardial infarction
Excessive immune‐mediated inflammatory reaction plays a deleterious role in ventricular remodelling after myocardial infarction (MI). Interleukin (IL)‐38 is a newly characterized cytokine of the IL‐1 family and has been reported to exert a protective effect in some autoimmune diseases. However, its...
שמור ב:
| הוצא לאור ב: | J Cell Mol Med |
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| Main Authors: | , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
John Wiley and Sons Inc.
2019
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6933378/ https://ncbi.nlm.nih.gov/pubmed/31746138 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.14741 |
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