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Experimental atopic dermatitis is dependent on the TWEAK/Fn14 signaling pathway
Tumor necrosis factor (TNF)‐like weak inducer of apoptosis (TWEAK) acts through its receptor fibroblast growth factor inducible 14 (Fn14), and participates in skin inflammation. Both TWEAK and Fn14 are highly expressed in skin lesions of patients with atopic dermatitis. The purpose of this study was...
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| 出版年: | Clin Exp Immunol |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6904660/ https://ncbi.nlm.nih.gov/pubmed/31515807 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cei.13373 |
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