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Loss of ACOT7 potentiates seizures and metabolic dysfunction
Neurons uniquely antagonize fatty acid utilization by hydrolyzing the activated form of fatty acids, long chain acyl-CoAs, via the enzyme acyl-CoA thioesterase 7, Acot7. The loss of Acot7 results in increased fatty acid utilization in neurons and exaggerated stimulus-evoked behavior such as an incre...
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| Publicat a: | Am J Physiol Endocrinol Metab |
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| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6879868/ https://ncbi.nlm.nih.gov/pubmed/31039008 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00537.2018 |
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