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Thymosin β4 promotes autophagy and repair via HIF-1α stabilization in chronic granulomatous disease
Chronic granulomatous disease (CGD) is a genetic disorder of the NADPH oxidase characterized by increased susceptibility to infections and hyperinflammation associated with defective autophagy and increased inflammasome activation. Herein, we demonstrate that thymosin β4 (Tβ4), a g-actin sequesterin...
Tallennettuna:
| Julkaisussa: | Life Sci Alliance |
|---|---|
| Päätekijät: | , , , , , , , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Life Science Alliance LLC
2019
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6851533/ https://ncbi.nlm.nih.gov/pubmed/31719116 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.26508/lsa.201900432 |
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