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Redox-mediated programed death of myocardial cells after cardiac arrest and cardiopulmonary resuscitation

Besides the fact that prolonged whole-body ischemia causes tissue and organ injury during cardiac arrest, additional damage occurs after the restoration of spontaneous circulation, during which the reperfusion activates a host of intracellular responses. These responses may lead to an increased thre...

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Détails bibliographiques
Publié dans:Redox Rep
Auteurs principaux: Chalkias, Athanasios, Xanthos, Theodoros
Format: Artigo
Langue:Inglês
Publié: Taylor & Francis 2013
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6837548/
https://ncbi.nlm.nih.gov/pubmed/22333162
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1179/1351000212Y.0000000002
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