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BTXA regulates the epithelial–mesenchymal transition and autophagy of keloid fibroblasts via modulating miR-1587/miR-2392 targeted ZEB2

Keloids are very resistant to treatment in dermatology and plastic surgical practice. The present study aimed to explore the underlying mechanism of botulinum toxin A (BTXA) treated human skin keloid fibroblasts (HSFBs) proving some new insights into keloids treatment. Expression of miR-1587 and miR...

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Dettagli Bibliografici
Pubblicato in:Biosci Rep
Autori principali: Hou, Zhanying, Fan, Feixiang, Liu, Po
Natura: Artigo
Lingua:Inglês
Pubblicazione: Portland Press Ltd. 2019
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6822502/
https://ncbi.nlm.nih.gov/pubmed/31652445
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BSR20190679
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