Antigenic Pressure on H3N2 Influenza Virus Drift Strains Imposes Constraints on Binding to Sialylated Receptors but Not Phosphorylated Glycans

H3N2 strains of influenza A virus emerged in humans in 1968 and have continued to circulate, evolving in response to human immune pressure. During this process of “antigenic drift,” viruses have progressively lost the ability to agglutinate erythrocytes of various species and to replicate efficientl...

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Bibliographische Detailangaben
Veröffentlicht in:J Virol
Hauptverfasser: Byrd-Leotis, Lauren, Gao, Chao, Jia, Nan, Mehta, Akul Y., Trost, Jessica, Cummings, Sandra F., Heimburg-Molinaro, Jamie, Cummings, Richard D., Steinhauer, David A.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Society for Microbiology 2019
Schlagworte:
Virus-Cell Interactions
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6819937/
https://ncbi.nlm.nih.gov/pubmed/31484755
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.01178-19
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