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Muscle metabolic reprogramming underlies the resistance of liver fatty acid–binding protein (LFABP)-null mice to high-fat feeding–induced decline in exercise capacity
Liver fatty acid-binding protein (LFABP) binds long-chain fatty acids with high affinity and is abundantly expressed in the liver and small intestine. Although LFABP is thought to function in intracellular lipid trafficking, studies of LFABP-null (LFABP(−/−)) mice have also indicated a role in regul...
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| Publicado no: | J Biol Chem |
|---|---|
| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6802512/ https://ncbi.nlm.nih.gov/pubmed/31451493 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA118.006684 |
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