Attenuation of Ischemia-Induced Cellular and Behavioral Deficits by X Chromosome-Linked Inhibitor of Apoptosis Protein Overexpression in the Rat Hippocampus

Transient forebrain ischemia produced by four-vessel occlusion (4-VO) triggers the delayed death of CA1 neurons in the hippocampus, resulting in behavioral deficits of spatial learning performance. We demonstrate that CA1 neuronal loss induced by 4-VO (12 min) is preceded by a selective and marked e...

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Pubblicato in:J Neurosci
Autori principali: Xu, Daigen, Bureau, Yves, McIntyre, Dan C., Nicholson, Donald W., Liston, Peter, Zhu, Yanxia, Fong, Wei Gin, Crocker, Stephen J., Korneluk, Robert G., Robertson, George S.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Society for Neuroscience 1999
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Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6782658/
https://ncbi.nlm.nih.gov/pubmed/10366635
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.19-12-05026.1999
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