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The Drosophila β-Amyloid Precursor Protein Homolog Promotes Synapse Differentiation at the Neuromuscular Junction
Although abnormal processing of β-amyloid precursor protein (APP) has been implicated in the pathogenic cascade leading to Alzheimer’s disease, the normal function of this protein is poorly understood. To gain insight into APP function, we used a molecular-genetic approach to manipulate the structur...
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| 出版年: | J Neurosci |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Society for Neuroscience
1999
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6782486/ https://ncbi.nlm.nih.gov/pubmed/10479682 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.19-18-07793.1999 |
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