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血管紧张素Ⅱ通过上调富含半胱氨酸蛋白61表达诱导HEK293T细胞凋亡
OBJECTIVE: To investigate the role of Cyr61 in angiotensin Ⅱ (AngⅡ)-induced functional changes in HEK293 cells and explore the mechanism. METHODS: Cyr61 knockdown in cultured HEK293T cells was achieved by transfection of the cells with CRISPR/Cas9 KO plasmid. The changes in apoptosis and expression...
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| Pubblicato in: | Nan Fang Yi Ke Da Xue Xue Bao |
|---|---|
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
南方医科大学学报编辑部
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6765560/ https://ncbi.nlm.nih.gov/pubmed/31340914 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12122/j.issn.1673-4254.2019.07.10 |
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