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The myristoylated alanine-rich C kinase substrate (MARCKS) differentially regulates kinase interacting with stathmin (KIS) in vascular smooth muscle and endothelial cells and potentiates intimal hyperplasia formation
OBJECTIVE: Restenosis limits the durability of all cardiovascular reconstructions. Vascular smooth muscle cell (VSMC) proliferation drives this process, but an intact, functional endothelium is necessary for vessel patency. Current strategies to prevent restenosis employ anti-proliferative agents th...
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| Publicado no: | J Vasc Surg |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6765458/ https://ncbi.nlm.nih.gov/pubmed/30929966 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jvs.2018.12.022 |
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