β-Amyloid Induces Neuronal Apoptosis Via a Mechanism that Involves the c-Jun N-Terminal Kinase Pathway and the Induction of Fas Ligand

Elevated levels of β-Amyloid (Aβ) are present in the brains of individuals with either the sporadic or familial form of Alzheimer's disease (AD), and the deposition of Aβ within the senile plaques that are a hallmark of AD is thought to be a primary cause of the cognitive dysfunction that occur...

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Bibliografiske detaljer
Udgivet i:J Neurosci
Main Authors: Morishima, Yoshiyuki, Gotoh, Yukiko, Zieg, Janine, Barrett, Tamera, Takano, Hiromichi, Flavell, Richard, Davis, Roger J., Shirasaki, Yasufumi, Greenberg, Michael E.
Format: Artigo
Sprog:Inglês
Udgivet: Society for Neuroscience 2001
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ARTICLE
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6762892/
https://ncbi.nlm.nih.gov/pubmed/11567045
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.21-19-07551.2001
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