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Activation of Bcl-2-Associated Death Protein and Counter-Response of Akt within Cell Populations during Seizure-Induced Neuronal Death

Bcl-2 family gene products are critical to the integration of cell death stimuli that target the mitochondrion. Proapoptotic BAD (Bcl-2-associated death protein) has been shown to dissociate from its sequestered site with the molecular chaperone protein 14-3-3 and displace proapoptotic BAX (Bcl-2-as...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Henshall, David C., Araki, Tomohiro, Schindler, Clara K., Lan, Jing-Quan, Tiekoter, Kenneth L., Taki, Waro, Simon, Roger P.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2002
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6757765/
https://ncbi.nlm.nih.gov/pubmed/12351720
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-19-08458.2002
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